There is a simple but highly impressive experiment that ostensibly could be done at home though in reality more easily in a well maintained laboratory. Let us assume we have access to the latter.
Add about 1mL of a biological buffer at pH 7 to a transparent plastic tube. The buffer solution is clear. Now add about 50 microlitres of a standard iron solution (28 mM) to the buffer, mix thoroughly and watch. Slowly the clear buffer solution begins to turn orange demonstrating the precipitation of iron (III) hydroxide, you have formed rust. Actually you have established what is called a redox cycle between iron (II) hydroxide (colourless) and iron (III) hydroxide (rust) and this cycle is catalysed or maintained by sunlight (UV radiation). The latter through the formation in the buffer solution of the superoxide radical anion slowly converts the insoluble iron (III) hydroxide precipitate (rust) to soluble iron (II) (colourless). The equilibrium between these two oxidation states of iron (Fe(II) and Fe(III)) favours iron (III) and hence the solution remains orange to the naked eye.
Stay with me on this, it is about to get interesting.
Now add about 1 microlitre of a standard aluminium solution (37mM) to the orange-coloured buffer, mix well and watch. Within a relatively short period of time the orange solution becomes clear. What you observe is the dissolution of the orange iron (III) hydroxide precipitate by aluminium (III). The presence of only a trace amount of aluminium (Al3+) has catalysed the formation of iron (II) from iron (III). Aluminium pushes the equilibrium to favour the presence of iron (II). How does aluminium, which only has one oxidation state (Al(III)) in biology, promote the reduction of iron (III) to iron (II)?
Aluminium is described as a pro-oxidant and this property of aluminium has been described many, many times without any attribution of a mechanism. I ‘mused’ about this long and hard for several years and eventually in 2004 I proposed a mechanism whereby aluminium may act as a pro-oxidant. I proposed a reaction between aluminium (Al3+) and the superoxide radical anion to form an aluminium superoxide semi-reduced radical ion, AlO2·2+. While we do not as yet have a method to directly identify the presence of this ion in solution, in any biological milieu, its existence and mode of action has been proven using computational chemistry. I am not too modest to say that AlO2·2+ is a beautiful solution to what was thought an intractable problem. I am pleased to know that many scientists agree with me and the original paper has been cited hundreds of times.
The practical consequences, indeed toxicological consequences, of aluminium acting as a pro-oxidant are myriad and potentially critical to the role of aluminium in human disease. Anywhere in the body where oxidative damage is happening, that damage will be increased many, many times by the presence of only trace amounts of aluminium. One example of this is oxidative damage in the vicinity of senile plaques in Alzheimer’s disease.
PS If you cannot access the seminal papers mentioned herein then drop me an email (drchrisexley@protonmail.com) and I will send you the pdf.
I do not think the we know everything these big pharma clowns add to vaccines (and drugs, for that matter). It's always been an "industry" secret or a "proprietary" secret. If they weren't adding a bunch of debilitating garbage, then there would be no harm in revealing the entire contents.
That’s why when we complained about mercury in vaccines, they replaced it with something even worse.